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SN11 SHORT NOTE 5M

Write a short note on the important clinical signs in respiratory disease.

General Medicine · Final Professional

Added Sounds

SignMechanismCauses
WHEEZE (rhonchi)Musical continuous sound from turbulent flow through NARROWED intrathoracic airways; typically EXPIRATORYPOLYPHONIC and widespread: ASTHMA, COPD, "cardiac asthma". MONOPHONIC and FIXED: LOCALISED OBSTRUCTION — tumour, foreign body, mucus plug — always warrants investigation
STRIDORHarsh, high-pitched, predominantly INSPIRATORY sound from EXTRATHORACIC UPPER AIRWAY obstructionForeign body, epiglottitis, croup, ANAPHYLAXIS, vocal cord palsy, laryngeal tumour, tracheal stenosis, retrosternal goitre. ALWAYS AN EMERGENCY
CRACKLESDiscontinuous popping from sudden opening of collapsed airways or air through secretionsFINE, END-INSPIRATORY, "VELCRO", BASAL: FIBROSIS/ILD, pulmonary OEDEMA. COARSE, EARLY-INSPIRATORY, altering with coughing: BRONCHIECTASIS, secretions, pneumonia
PLEURAL RUBCreaking; INSPIRATORY AND EXPIRATORY; disappears on breath-holdingPleurisy — pneumonia, pulmonary infarction, TB, connective tissue disease, uraemia
BRONCHIAL BREATHINGHarsh, with a GAP between inspiration and expiration, expiration ≥ inspirationCONSOLIDATION (classic); above a pleural effusion; over collapse with a patent bronchus; over a large cavity (amphoric)

Voice-conducted Signs

All depend on one principle: solid consolidated lung conducts sound BETTER than air-filled lung; fluid and air in the pleural space conduct WORSE.

SignTestIncreased inDecreased in
VOCAL FREMITUSPalpate while the patient says "ninety-nine"ConsolidationEffusion, pneumothorax, collapse with obstructed bronchus, pleural thickening
VOCAL RESONANCEAuscultate while saying "ninety-nine"ConsolidationAs above
WHISPERING PECTORILOQUYAuscultate while the patient WHISPERS — whispered sounds become clearly audibleCONSOLIDATION (most sensitive voice sign); above an effusion—
EGOPHONY ("goat voice")Patient says "eeee" — heard as a nasal "aaaay" (E-to-A change)Consolidation, and classically at the UPPER LEVEL of an EFFUSION—

Other Signs

  • BARREL CHEST — increased anteroposterior diameter, horizontal ribs, increased subcostal angle, from chronic HYPERINFLATION — COPD/emphysema, chronic severe asthma, cystic fibrosis; with hyperresonance and loss of cardiac and hepatic dullness
  • TRACHEAL DEVIATION — TOWARDS the lesion: collapse, fibrosis, pneumonectomy (volume loss). AWAY: large effusion, TENSION pneumothorax, large mass. A displaced trachea in a breathless hypotensive patient means tension pneumothorax until proved otherwise
  • CLUBBING — respiratory causes: bronchogenic carcinoma, mesothelioma, bronchiectasis, lung abscess, empyema, cystic fibrosis, ILD, asbestosis. NOT caused by uncomplicated COPD or asthma — its presence in a COPD patient demands a search for cancer, bronchiectasis or abscess
  • HOOVER'S SIGN — paradoxical inward movement of the lower ribs on inspiration, from a flattened diaphragm
  • PULSUS PARADOXUS — inspiratory fall in systolic pressure > 10 mmHg; in respiratory practice indicates acute severe asthma or COPD
  • ASTERIXIS — CO2 retention (type II respiratory failure)
  • SUCCUSSION SPLASH — HYDROPNEUMOTHORAX

Putting the Signs Together

ConditionExpansionPercussionBreath soundsFremitus/resonanceTrachea
CONSOLIDATIONReducedDULLBRONCHIALINCREASEDCentral
PLEURAL EFFUSIONReducedSTONY DULLABSENTDECREASEDAWAY (if large)
PNEUMOTHORAXReducedHYPERRESONANTABSENTDECREASEDAWAY (tension)
COLLAPSEReducedDullABSENTDECREASEDTOWARDS
FIBROSISReducedDull or normalFine cracklesIncreased or normalTOWARDS (if localised)
COPDReduced, symmetricalHYPERRESONANTReduced, prolonged expiration, wheezeReducedCentral
CLINICAL PEARL

Percussion and the trachea together separate the four classic unilateral pathologies in seconds. Dull + trachea AWAY = effusion. Dull + trachea TOWARDS = collapse or fibrosis. Hyperresonant + trachea AWAY = tension pneumothorax. Dull with BRONCHIAL breathing and INCREASED fremitus = consolidation. These four combinations account for the great majority of examination cases and of real emergencies, and they require no equipment at all.

All questions in Respiratory System

  1. Q1Discuss the aetiology, pathogenesis, clinical features, investigations and management of chronic obstructive pulmonary disease. ★
  2. Q2Discuss the aetiopathogenesis, clinical features, diagnosis and management of pulmonary tuberculosis. ★
  3. Q3Discuss the aetiopathogenesis, clinical features, investigations and management of bronchial asthma, including acute severe asthma. ★
  4. Q4Discuss the classification, aetiology, clinical features, severity assessment and management of pneumonia. ★
  5. Q5Discuss the definition, causes, pathophysiology, clinical features and management of acute respiratory distress syndrome. ★
  6. Q6Discuss the risk factors, clinical features, investigations and management of pulmonary thromboembolism. ★
  7. SN1Write a short note on pneumothorax and hydropneumothorax.
  8. SN2Write a short note on the clinical approach to acute dyspnoea.
  9. SN3Write a short note on pulmonary function tests.
  10. SN4Write a short note on respiratory failure.
  11. SN5Write a short note on pleural effusion.
  12. SN6Write a short note on eosinophilic lung disease — ABPA, hypersensitivity pneumonitis and tropical pulmonary eosinophilia.
  13. SN7Write a short note on bronchiectasis.
  14. SN8Write a short note on lung abscess.
  15. SN9Write a short note on haemoptysis.
  16. SN10Write a short note on interstitial lung disease.
  17. SN11Write a short note on the important clinical signs in respiratory disease.
  18. SN12Write a short note on bronchogenic carcinoma.

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