SN11
SHORT NOTE
5M
Write a short note on the important clinical signs in respiratory disease.
Added Sounds
| Sign | Mechanism | Causes |
|---|---|---|
| WHEEZE (rhonchi) | Musical continuous sound from turbulent flow through NARROWED intrathoracic airways; typically EXPIRATORY | POLYPHONIC and widespread: ASTHMA, COPD, "cardiac asthma". MONOPHONIC and FIXED: LOCALISED OBSTRUCTION — tumour, foreign body, mucus plug — always warrants investigation |
| STRIDOR | Harsh, high-pitched, predominantly INSPIRATORY sound from EXTRATHORACIC UPPER AIRWAY obstruction | Foreign body, epiglottitis, croup, ANAPHYLAXIS, vocal cord palsy, laryngeal tumour, tracheal stenosis, retrosternal goitre. ALWAYS AN EMERGENCY |
| CRACKLES | Discontinuous popping from sudden opening of collapsed airways or air through secretions | FINE, END-INSPIRATORY, "VELCRO", BASAL: FIBROSIS/ILD, pulmonary OEDEMA. COARSE, EARLY-INSPIRATORY, altering with coughing: BRONCHIECTASIS, secretions, pneumonia |
| PLEURAL RUB | Creaking; INSPIRATORY AND EXPIRATORY; disappears on breath-holding | Pleurisy — pneumonia, pulmonary infarction, TB, connective tissue disease, uraemia |
| BRONCHIAL BREATHING | Harsh, with a GAP between inspiration and expiration, expiration ≥ inspiration | CONSOLIDATION (classic); above a pleural effusion; over collapse with a patent bronchus; over a large cavity (amphoric) |
Voice-conducted Signs
All depend on one principle: solid consolidated lung conducts sound BETTER than air-filled lung; fluid and air in the pleural space conduct WORSE.
| Sign | Test | Increased in | Decreased in |
|---|---|---|---|
| VOCAL FREMITUS | Palpate while the patient says "ninety-nine" | Consolidation | Effusion, pneumothorax, collapse with obstructed bronchus, pleural thickening |
| VOCAL RESONANCE | Auscultate while saying "ninety-nine" | Consolidation | As above |
| WHISPERING PECTORILOQUY | Auscultate while the patient WHISPERS — whispered sounds become clearly audible | CONSOLIDATION (most sensitive voice sign); above an effusion | — |
| EGOPHONY ("goat voice") | Patient says "eeee" — heard as a nasal "aaaay" (E-to-A change) | Consolidation, and classically at the UPPER LEVEL of an EFFUSION | — |
Other Signs
- BARREL CHEST — increased anteroposterior diameter, horizontal ribs, increased subcostal angle, from chronic HYPERINFLATION — COPD/emphysema, chronic severe asthma, cystic fibrosis; with hyperresonance and loss of cardiac and hepatic dullness
- TRACHEAL DEVIATION — TOWARDS the lesion: collapse, fibrosis, pneumonectomy (volume loss). AWAY: large effusion, TENSION pneumothorax, large mass. A displaced trachea in a breathless hypotensive patient means tension pneumothorax until proved otherwise
- CLUBBING — respiratory causes: bronchogenic carcinoma, mesothelioma, bronchiectasis, lung abscess, empyema, cystic fibrosis, ILD, asbestosis. NOT caused by uncomplicated COPD or asthma — its presence in a COPD patient demands a search for cancer, bronchiectasis or abscess
- HOOVER'S SIGN — paradoxical inward movement of the lower ribs on inspiration, from a flattened diaphragm
- PULSUS PARADOXUS — inspiratory fall in systolic pressure > 10 mmHg; in respiratory practice indicates acute severe asthma or COPD
- ASTERIXIS — CO2 retention (type II respiratory failure)
- SUCCUSSION SPLASH — HYDROPNEUMOTHORAX
Putting the Signs Together
| Condition | Expansion | Percussion | Breath sounds | Fremitus/resonance | Trachea |
|---|---|---|---|---|---|
| CONSOLIDATION | Reduced | DULL | BRONCHIAL | INCREASED | Central |
| PLEURAL EFFUSION | Reduced | STONY DULL | ABSENT | DECREASED | AWAY (if large) |
| PNEUMOTHORAX | Reduced | HYPERRESONANT | ABSENT | DECREASED | AWAY (tension) |
| COLLAPSE | Reduced | Dull | ABSENT | DECREASED | TOWARDS |
| FIBROSIS | Reduced | Dull or normal | Fine crackles | Increased or normal | TOWARDS (if localised) |
| COPD | Reduced, symmetrical | HYPERRESONANT | Reduced, prolonged expiration, wheeze | Reduced | Central |
CLINICAL PEARL
Percussion and the trachea together separate the four classic unilateral pathologies in seconds. Dull + trachea AWAY = effusion. Dull + trachea TOWARDS = collapse or fibrosis. Hyperresonant + trachea AWAY = tension pneumothorax. Dull with BRONCHIAL breathing and INCREASED fremitus = consolidation. These four combinations account for the great majority of examination cases and of real emergencies, and they require no equipment at all.
All questions in Respiratory System
- Q1Discuss the aetiology, pathogenesis, clinical features, investigations and management of chronic obstructive pulmonary disease. ★
- Q2Discuss the aetiopathogenesis, clinical features, diagnosis and management of pulmonary tuberculosis. ★
- Q3Discuss the aetiopathogenesis, clinical features, investigations and management of bronchial asthma, including acute severe asthma. ★
- Q4Discuss the classification, aetiology, clinical features, severity assessment and management of pneumonia. ★
- Q5Discuss the definition, causes, pathophysiology, clinical features and management of acute respiratory distress syndrome. ★
- Q6Discuss the risk factors, clinical features, investigations and management of pulmonary thromboembolism. ★
- SN1Write a short note on pneumothorax and hydropneumothorax.
- SN2Write a short note on the clinical approach to acute dyspnoea.
- SN3Write a short note on pulmonary function tests.
- SN4Write a short note on respiratory failure.
- SN5Write a short note on pleural effusion.
- SN6Write a short note on eosinophilic lung disease — ABPA, hypersensitivity pneumonitis and tropical pulmonary eosinophilia.
- SN7Write a short note on bronchiectasis.
- SN8Write a short note on lung abscess.
- SN9Write a short note on haemoptysis.
- SN10Write a short note on interstitial lung disease.
- SN11Write a short note on the important clinical signs in respiratory disease.
- SN12Write a short note on bronchogenic carcinoma.