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Describe herpes zoster oticus.

ENT · 3rd Professional

Nature and Presentation

Herpes zoster oticus (RAMSAY hunt syndrome) is reactivation of latent varicella-zoster virus in the geniculate ganglion of the facial nerve, producing the triad of otalgia, vesicles in the ear, and lower motor neurone facial palsy.

  • The virus lies dormant in the sensory ganglion after chickenpox, and reactivates when cell-mediated immunity declines — with age, immunosuppression, HIV, malignancy, diabetes and stress. Ramsay Hunt in a young patient should prompt HIV testing
  • The vesicles appear in the concha, external canal and postauricular skin — the small area of skin supplied by the sensory branch of the facial nerve, which is why this apparently obscure sensory territory matters

Management and Prognosis

  • The prognosis IS significantly worse than bell’S palsy, and this is the central clinical fact: complete recovery occurs in only 50 to 60% against some 85 to 90% for Bell’s palsy, and a complete palsy at presentation carries a much worse outlook
  • Treatment must begin early — ideally within 72 hours, and the benefit falls off sharply with delay.
  • Antiviral — aciclovir 800 mg five times daily, or valaciclovir or famciclovir, for 7 to 10 days; intravenously in severe or immunosuppressed cases
  • Corticosteroid — prednisolone 1 mg/kg tapering over 10 to 14 days.
  • The combination of antiviral and steroid is standard in Ramsay Hunt, whereas steroid alone suffices in Bell’s palsy
  • Analgesia, which is frequently the patient’s main concern, and may require gabapentin, pregabalin or amitriptyline for the neuropathic component
  • Eye care IS the urgent priority and must not be left to the patient — lubricant drops by day, ointment and taping at night, a protective shield, and tarsorrhaphy if the cornea is threatened. The risk is greater than in Bell’s palsy.
  • Vestibular sedatives for vertigo, used briefly only
  • Physiotherapy and facial exercises once recovery begins; and surgical decompression, which is controversial and of unproven benefit
  • Follow up with House-Brackmann grading and audiometry
  • Complications — post-herpetic neuralgia.
  • Prevention — the zoster vaccine reduces the incidence of shingles and of post-herpetic neuralgia in the elderly
  • Otalgia precedes the rash, so the patient may present before there is anything visible in the canal
  • Eighth nerve involvement distinguishes it from Bell palsy, giving deafness, tinnitus and vertigo alongside the facial weakness
  • Recovery is only fifty to sixty per cent, against eighty-five to ninety for Bell palsy.
  • Treat within seventy-two hours; the benefit of antiviral therapy falls off sharply after that

All questions in Diseases of the External Ear

  1. Q1Describe otitis externa with its types, causes and management. ★
  2. Q2Describe malignant otitis externa. ★
  3. Q3Describe wax and foreign bodies in the ear with their management. ★
  4. Q4Describe the tumours of the external ear. ★
  5. Q5Describe the injuries of the pinna, external canal and tympanic membrane. ★
  6. SN1Describe otomycosis with its management. ★
  7. SN2Describe furuncle of the external auditory canal. ★
  8. SN3Describe perichondritis of the pinna and relapsing polychondritis. ★
  9. SN4Describe herpes zoster oticus. ★
  10. SN5Describe keratosis obturans and external canal cholesteatoma. ★
  11. SN6Describe exostosis and osteoma of the external auditory canal. ★
  12. SN7Describe myringitis and acquired stenosis of the external canal.

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