Orthopaedics
Trauma and orthopaedics for the Indian MBBS final-year exam — fractures, dislocations, bone and joint infection, tumours, the spine, arthritis, paediatric and regional orthopaedics. Full-length explanation-first answers with diagrams.
Definition & Surgical Importance
A fracture of the neck of the femur is an intracapsular fracture occurring between the femoral head and the intertrochanteric line. It is common in the elderly osteoporotic patient after a trivial fall, and in the young only after high-energy trauma. Its importance lies in the precarious blood supply of the femoral head, which is largely intracapsular — hence a high incidence of avascular necrosis (AVN) and non-union.
Blood Supply of the Femoral Head (why it matters)
Three sources supply the head: (1) the retinacular vessels from the medial and lateral circumflex femoral arteries (the dominant supply, running in the capsule up the neck — torn in displaced fractures); (2) the artery of the ligamentum teres (foveal artery — minor, often absent in the elderly); and (3) intra-osseous vessels from the shaft (interrupted at the fracture line). A displaced intracapsular fracture therefore strips the head of its blood supply, explaining AVN.
💡The more displaced and the more vertical the fracture line, the higher the shear force and the poorer the prognosis for union and head viability.Garden classification of intracapsular neck-of-femur fractures based on displacement on the AP radiograph. Classification
Garden classification (by displacement): Type I incomplete/impacted (valgus); Type II complete but undisplaced; Type III complete with partial displacement (trabeculae malaligned); Type IV complete with full displacement (head free, trabeculae parallel again). Pauwels classification (by the angle of the fracture line to the horizontal): Type I <30°, Type II 30–50°, Type III >50° — the more vertical, the greater the shear and the higher the failure rate. Anatomically: subcapital, transcervical and basicervical.
Feature Intracapsular (neck) Extracapsular (trochanteric) Blood supply Precarious — AVN common Rich — AVN rare, unites well Typical age Elderly & young high-energy Very elderly, osteoporotic Limb posture Shortened, externally rotated (~45°) Shortened, externally rotated (~90°) Main problems AVN, non-union Malunion (coxa vara), blood loss Clinical Features
The patient is unable to bear weight after a fall; the limb is shortened, adducted and externally rotated (about 45°, less than the 90° of trochanteric fractures because the intact capsule limits rotation). There is pain in the groin, tenderness over Scarpa's triangle, and pain on axial (heel) percussion. An impacted (Garden I) fracture may still allow limited weight-bearing — a trap.
⚠️An elderly patient with hip/groin/knee pain and inability to bear weight after even a trivial fall has a hip fracture until proven otherwise. If radiographs are normal but suspicion is high, obtain MRI (or repeat films/CT) to exclude an occult fracture.Investigations
AP pelvis and lateral (cross-table) radiographs of the affected hip. Shenton's line is broken. Look for the fracture line and disruption of the trabecular pattern. MRI is the investigation of choice for a suspected occult fracture. Routine pre-operative work-up (bloods, ECG, chest radiograph) is essential as most patients are elderly with comorbidities.
Management
Undisplaced (Garden I & II): internal fixation with three cannulated cancellous screws in an inverted-triangle configuration. Displaced (Garden III & IV): treatment depends on age and physiological status. In the young adult the head must be salvaged — emergency closed/open reduction and internal fixation within a few hours to reduce AVN risk. In the elderly, arthroplasty is preferred: hemiarthroplasty (bipolar/unipolar) for the frail, low-demand patient, and total hip replacement for the fit, active, independent patient (better function, lower re-operation). Surgery is ideally performed within 48 hours to reduce mortality and complications. Early mobilisation, DVT prophylaxis and osteoporosis treatment complete the plan.
💡Fix the young, replace the old. Salvage the head with urgent fixation in the young; in the elderly, a displaced intracapsular fracture is best treated with arthroplasty to allow immediate weight-bearing and avoid the high re-operation rate of fixation.Complications
Early: general complications of recumbency in the elderly (chest infection, DVT/PE, pressure sores, delirium), and high one-year mortality. Late: avascular necrosis (up to 30% of displaced fractures, may appear 1–3 years later), non-union, and secondary osteoarthritis.
🔑KEY POINTS TO REMEMBER- Intracapsular fracture → precarious blood supply → high AVN & non-union.
- Garden I–II = undisplaced; III–IV = displaced. Pauwels grades by verticality.
- Limb shortened, adducted, externally rotated ~45°.
- Young + displaced = emergency ORIF (salvage head). Elderly + displaced = arthroplasty.
- Operate within 48 h; treat osteoporosis; give DVT prophylaxis.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition
An intertrochanteric (pertrochanteric) fracture is an extracapsular fracture of the proximal femur running between the greater and lesser trochanters. It is one of the commonest fractures in the elderly osteoporotic patient. Because the region is cancellous and richly vascular, union is the rule and avascular necrosis is rare — the problems here are blood loss, malunion (coxa vara) and implant failure, not non-union.
Mechanism & Clinical Features
Usually a fall on the greater trochanter in an elderly woman. The limb is markedly shortened and externally rotated (~90°) — more than in an intracapsular fracture because there is no intact capsule to restrain rotation. There is swelling, bruising and severe tenderness over the trochanter; the patient cannot bear weight. Significant blood loss into the thigh can cause hypovolaemia.
💡Compared with a neck fracture, the intertrochanteric limb is shortened and externally rotated to a greater degree (~90°) and shows more bruising — the fracture is outside the capsule.Classification
Boyd and Griffin and Evans classifications describe the pattern. The key clinical distinction is stable vs unstable. Stability depends on the integrity of the posteromedial cortex (calcar): comminution of the posteromedial fragment, a reverse oblique line, or subtrochanteric extension makes the fracture unstable and prone to collapse into varus.
Stable pattern Unstable pattern Posteromedial cortex Intact / reconstructable Comminuted (calcar loss) Fracture line Simple two-part Reverse oblique / subtroch. extension Tendency Resists collapse after reduction Collapses into coxa vara Preferred implant DHS acceptable Cephalomedullary nail (PFN/PFNA) Investigations
AP pelvis and lateral hip radiographs demonstrate the fracture and its comminution. Assess the posteromedial cortex and any reverse-oblique component for stability. Full pre-operative assessment and correction of anaemia/dehydration are important given the elderly population and blood loss.
Management
Treatment is almost always operative to allow early mobilisation and avoid the lethal complications of recumbency. Options:
• Dynamic hip screw (DHS) — a lag screw in the head connected to a side-plate on the shaft. It allows controlled dynamic impaction at the fracture, ideal for stable patterns.
• Cephalomedullary nail (PFN / PFNA / Gamma nail) — an intramedullary device with a cephalic screw/blade; the shorter lever arm and load-sharing make it the implant of choice for unstable, reverse-oblique and subtrochanteric patterns.
• Arthroplasty is reserved for selected cases (e.g. severe comminution with poor bone or pre-existing arthritis). Surgery should ideally occur within 48 hours, with DVT prophylaxis, early weight-bearing and osteoporosis management.⚠️The dreaded technical failure is lag-screw cut-out through the femoral head. It is minimised by achieving a good reduction and placing the screw central and deep in the head with a low tip–apex distance (TAD < 25 mm).Subtrochanteric Extension
A fracture involving the region within 5 cm below the lesser trochanter is subtrochanteric. This is a high-stress area of predominantly cortical bone subjected to large compressive (medial) and tensile (lateral) forces, so it is prone to varus malunion and implant failure. A long cephalomedullary nail is the implant of choice. An ‘atypical’ subtrochanteric fracture (transverse, lateral cortical beaking) should raise suspicion of prolonged bisphosphonate use.
Peri-operative Care & Rehabilitation
Because these are frail, elderly patients, outcome depends as much on medical co-management as on the surgery: correction of anaemia and dehydration, analgesia, delirium prevention, pressure-area care, DVT prophylaxis and early nutritional support. Ortho-geriatric shared care and surgery within 48 hours reduce mortality. Post-operatively the aim is immediate weight-bearing as tolerated with physiotherapy, and secondary fracture prevention with calcium, vitamin D and bone-protection therapy.
Complications
General complications of immobility and high one-year mortality in the frail elderly. Mechanical: coxa vara / malunion from collapse of an unstable fracture, lag-screw cut-out, and implant breakage. Non-union and AVN are uncommon because of the good blood supply.
Extracapsular — avascular necrosis is rare, unlike neck fractures. 🔑KEY POINTS TO REMEMBER- Extracapsular → good blood supply → unites well; AVN & non-union rare.
- Limb shortened + externally rotated ~90° (more than intracapsular).
- Stability hinges on the posteromedial cortex (calcar).
- Stable → DHS; unstable / reverse-oblique / subtrochanteric → cephalomedullary nail.
- Avoid cut-out: good reduction + central deep screw + TAD < 25 mm.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition & Importance
A fracture of the shaft of the femur involves the diaphysis between the subtrochanteric region and the supracondylar region. The femur is the largest and strongest bone, so a shaft fracture usually implies high-energy trauma (road-traffic accident, fall from height) in a young adult, or a trivial injury on pathological/osteoporotic bone. It is important for two reasons: substantial blood loss (1–1.5 L into the thigh, more if bilateral or open) that can cause hypovolaemic shock, and the risk of fat embolism syndrome.
Deforming Forces
The powerful thigh muscles displace the fragments predictably. The proximal fragment is flexed (iliopsoas), abducted (glutei) and externally rotated; the distal fragment is pulled into adduction and varus by the adductors and flexed posteriorly by gastrocnemius (a danger to the popliteal vessels in supracondylar extensions). Understanding these forces guides reduction.
Characteristic deforming muscle pull on femoral shaft fragments — the basis of the typical deformity. Clinical Features
Severe pain, swelling and deformity of the thigh with shortening and inability to bear weight; abnormal mobility and crepitus. Because of the energy involved, assess for shock and always look for associated injuries — ipsilateral hip/neck-of-femur fracture, knee ligament injury, and (critically) distal neurovascular status.
⚠️Always examine the ipsilateral hip and knee. A neck-of-femur fracture is missed in up to 5% of femoral shaft fractures — dedicated hip radiographs/CT are mandatory. Also document distal pulses and sensation before and after any manipulation.Investigations
AP and lateral radiographs of the whole femur including the hip and knee joints — never image the shaft alone. Assess comminution, segmental patterns and pathological features. Trauma work-up (FAST/CT) as indicated by the mechanism.
Management
First aid / resuscitation: ATLS principles, control haemorrhage, splint the limb (Thomas splint) for pain relief and to reduce blood loss and fat embolism, and give analgesia.
Definitive: in adults the treatment of choice is closed intramedullary interlocking nailing — it is load-sharing, permits early mobilisation and gives high union rates. Antegrade or retrograde nails are used depending on the fracture and associated injuries. Plating or an external fixator (as damage control in the unstable polytrauma patient or open fracture) are alternatives. In children, treatment is largely conservative/age-dependent — Pavlik harness or hip spica in infants, and titanium elastic nails or submuscular plating in older children.💡An interlocking intramedullary nail is the gold standard for an adult femoral shaft fracture: it controls length, alignment and rotation, is load-sharing, and allows early mobilisation.Complications
Early: hypovolaemic shock, fat embolism syndrome, and (rarely) vascular or nerve injury. Late: delayed/non-union, malunion (shortening, malrotation, angulation), knee stiffness from quadriceps adhesions, and infection after open injury or surgery. Refracture may follow premature implant removal.
🔑KEY POINTS TO REMEMBER- High-energy injury in the young; watch for shock and fat embolism.
- Proximal fragment flexed/abducted; distal fragment adducted (varus) & flexed.
- Always X-ray the whole femur with hip and knee; exclude ipsilateral neck fracture.
- Splint with Thomas splint; definitive Rx = closed interlocking IM nailing in adults.
- Children treated conservatively / age-appropriate (spica, elastic nails).
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition
A fracture of both bones of the leg denotes a fracture of the shafts of the tibia and fibula. The tibia is subcutaneous along its whole anteromedial border, so these are frequently open (compound) fractures with a high risk of infection, delayed union and the surgical emergency of acute compartment syndrome.
Mechanism
Two broad patterns: a direct blow (bumper injury, RTA) causing a transverse or comminuted fracture at the same level, often open; and an indirect (twisting) force producing a spiral fracture of the tibia and fibula at different levels. High-energy fractures carry the greatest risk of soft-tissue compromise.
Clinical Features
Pain, swelling, deformity and inability to bear weight; the sharp subcutaneous tibial crest may tent or pierce the skin. Examine the skin (open wound), the neurovascular status (dorsalis pedis and posterior tibial pulses, deep peroneal sensation in the first web space) and — repeatedly — for compartment syndrome.
The four fascial compartments of the leg. The anterior compartment is the most commonly affected; deep peroneal nerve sensation (first web space) is the earliest to be lost. Acute Compartment Syndrome — the critical complication
Bleeding and oedema within an unyielding osteofascial compartment raise the interstitial pressure until it exceeds capillary perfusion pressure, causing ischaemia of muscle and nerve. The cardinal sign is pain out of proportion to the injury, worsened by passive stretch of the compartment muscles. The classical late “5 Ps” (pain, paraesthesia, pallor, pulselessness, paralysis) are unreliable — pulses are usually present until very late. Diagnosis is clinical; if in doubt, measure compartment pressure (a within 30 mmHg of diastolic — i.e. low ΔP — indicates the need to decompress).
⚠️Compartment syndrome is a surgical emergency. The treatment is immediate open fasciotomy of all four compartments. Do not wait for pulselessness or paralysis — by then the muscle is already necrosing (leading to Volkmann-type contracture).Investigations
AP and lateral radiographs of the whole tibia including knee and ankle joints. Classify open fractures by the Gustilo–Anderson grade. Check haemoglobin and renal function; myoglobinuria suggests muscle necrosis.
Management
Closed, low-energy fractures that are stable and acceptably aligned may be treated in an above-knee cast (later a functional Sarmiento brace). Displaced, unstable or open fractures are treated operatively: closed intramedullary interlocking nailing is the workhorse for closed and low-grade open tibial shaft fractures; external fixation is preferred for severe open (Gustilo IIIB/C) injuries and damage control. Open fractures require urgent antibiotics, tetanus prophylaxis, thorough debridement and early soft-tissue cover. Fasciotomy is performed the moment compartment syndrome is suspected.
💡The subcutaneous position of the tibia makes these fractures commonly open and prone to delayed union; always search actively and repeatedly for compartment syndrome.Complications
Compartment syndrome and its sequela of ischaemic contracture; infection and osteomyelitis (especially in open fractures); delayed and non-union (the distal third has a poor blood supply); and malunion. The fibula, being non-weight-bearing, often unites uneventfully.
🔑KEY POINTS TO REMEMBER- Tibia is subcutaneous → high rate of open fractures & delayed union.
- Acute compartment syndrome = pain out of proportion + pain on passive stretch.
- Pulses present until late — do not wait for the 5 Ps.
- Treatment of compartment syndrome = emergency 4-compartment fasciotomy.
- Closed stable → cast; unstable/open → IM nail or external fixator; open → debride + cover.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition & Surgical Anatomy
Ankle injuries range from ligamentous sprains to fracture-dislocations of the ankle mortise — the socket formed by the distal tibia (plafond and medial malleolus), the fibula (lateral malleolus) and the strong syndesmotic ligaments that bind them, into which the talus sits. Stability depends on both the bony malleoli and the medial (deltoid), lateral and syndesmotic ligaments. A congruent mortise is essential — even a 1–2 mm lateral shift of the talus markedly reduces the contact area and predisposes to arthritis.
Mechanism & Classification
Most ankle fractures follow a rotational injury of the foot on the leg. Two classifications are used. The Danis–Weber classification is based on the level of the fibular fracture relative to the syndesmosis (simple and surgically useful). The Lauge-Hansen classification describes the mechanism by foot position and force direction (e.g. supination-external rotation, the commonest).
Weber type Level of fibular # Syndesmosis Typical stability A Below the syndesmosis Intact Usually stable B At the syndesmosis (spiral) May be injured Variable — depends on medial side C Above the syndesmosis Disrupted Unstable — often needs fixation 💡The higher the fibular fracture (Weber A→C), the more likely the syndesmosis is disrupted and the more unstable the ankle. A high fibular fracture with a medial injury is the Maisonneuve fracture — always examine the whole leg up to the knee.Clinical Features
Pain, swelling and inability to bear weight after a twisting injury; deformity in fracture-dislocations. Palpate for tenderness at both malleoli, the medial and lateral ligaments and — importantly — the proximal fibula (Maisonneuve). Assess the skin (fracture blisters, threatened skin over a displaced fragment) and the neurovascular status. The Ottawa ankle rules help decide who needs radiographs.
Investigations
AP, lateral and mortise-view radiographs of the ankle. Assess the talar shift, the medial clear space, and symmetry of the mortise. Radiograph the full length of the fibula if a Maisonneuve injury is suspected. CT helps in complex or posterior malleolar/pilon patterns.
Management
Stable, undisplaced fractures (most Weber A and undisplaced B without talar shift) are treated conservatively in a below-knee cast or walking boot with a period of protected weight-bearing. Unstable / displaced fractures (talar shift, displaced bimalleolar or trimalleolar, most Weber C) require open reduction and internal fixation to restore an anatomically congruent mortise — lateral malleolus with a plate and screws, medial malleolus with screws or a tension band, and syndesmotic stabilisation (screw or suture-button) where disrupted. A fracture-dislocation should be reduced urgently to relieve pressure on the skin and neurovascular structures.
⚠️An ankle fracture-dislocation tenting the skin is an emergency — reduce it immediately (before definitive imaging) to prevent skin necrosis and neurovascular compromise; splint and re-image afterwards.Ankle Sprains & the Ottawa Rules
The commonest ankle injury is a lateral ligament sprain from an inversion force, injuring the anterior talofibular ligament (ATFL) first, then the calcaneofibular ligament. Sprains are graded I (stretch), II (partial tear) and III (complete tear with instability). The Ottawa ankle rules guide the need for radiographs: image only if there is bony tenderness at the posterior edge or tip of either malleolus, tenderness at the navicular or base of the fifth metatarsal, or an inability to bear weight for four steps both immediately and in the department. Most sprains are managed with the PRICE/POLICE regimen (protection, optimal loading, rest, ice, compression, elevation) and early rehabilitation; complete tears with instability occasionally need repair.
Complications
Post-traumatic osteoarthritis (from an incongruent mortise or residual talar shift) is the commonest late problem; also stiffness, malunion, non-union of the medial malleolus, wound problems over the subcutaneous fibula, and chronic instability after ligamentous injuries.
Talar shift of even 1 mm markedly reduces contact area. 🔑KEY POINTS TO REMEMBER- Stability of the mortise depends on the malleoli + deltoid/syndesmotic ligaments.
- Weber A (below) → C (above) — higher fibular # = more syndesmotic disruption/instability.
- Even 1–2 mm talar shift halves contact area → arthritis; restore the mortise anatomically.
- Stable/undisplaced → cast; unstable/displaced → ORIF ± syndesmotic fixation.
- Examine the proximal fibula (Maisonneuve); reduce a fracture-dislocation urgently.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition & Mechanism
The patella is the largest sesamoid bone and part of the extensor mechanism of the knee. A fracture results either from a direct blow (dashboard injury, fall onto the knee — often comminuted/stellate) or from an indirect violent quadriceps contraction against resistance (a transverse fracture with distraction of the fragments).
Clinical Features
Pain, swelling and a tense haemarthrosis of the knee. A palpable gap between the fragments and, most importantly, an inability to perform a straight-leg raise / extend the knee against gravity indicates disruption of the extensor mechanism.
Classification
Fractures are described as undisplaced, transverse (displaced), comminuted/stellate, or vertical, and as osteochondral. The key questions are whether the fragments are separated (>2–3 mm) and whether the extensor mechanism is intact.
Tension-band principle: parallel K-wires with an anterior figure-of-eight wire convert the distracting quadriceps pull into compression across the fracture during flexion. Management
Undisplaced fractures with an intact extensor mechanism are treated conservatively in a cylinder cast / knee brace in extension for a few weeks with early quadriceps exercises. Displaced fractures (gap > 3 mm, articular step, or loss of active extension) require open reduction and internal fixation, classically tension-band wiring over two K-wires (which converts tensile quadriceps force into compression at the fracture). A severely comminuted lower pole may need partial patellectomy with reattachment of the patellar tendon; total patellectomy is a last resort as it weakens extension.
💡The two aims of treatment are to restore the extensor mechanism and to reconstruct the articular surface; tension-band wiring achieves both and permits early movement.⚠️A patient who cannot straight-leg-raise has a disrupted extensor mechanism until proven otherwise — even with an apparently minor fracture, this mandates surgical repair.Type Feature Management Undisplaced Extensor mechanism intact Cylinder cast Transverse displaced Extensor lag present Tension band wiring Comminuted Multiple fragments Partial / total patellectomy Vertical Usually undisplaced Conservative 🔑KEY POINTS TO REMEMBER- Direct blow → comminuted; indirect quads pull → transverse with a gap.
- Inability to extend the knee / do a straight-leg raise = extensor mechanism disruption.
- Undisplaced + intact extensor mechanism → cast in extension.
- Displaced → ORIF with tension-band wiring; comminuted pole → partial patellectomy.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition
The calcaneus is the most commonly fractured tarsal bone. The classic mechanism is a fall from a height onto the heels, driving the talus into the calcaneus (an axial compression injury). Fractures are intra-articular (involving the subtalar/posterior facet — the majority) or extra-articular.
⚠️A fall from height causing a calcaneal fracture demands a search for associated axial injuries: the contralateral calcaneus, tibial plateau, hip, and — classically — a compression fracture of the thoracolumbar spine. Examine the spine in every heel fracture.Clinical Features
A painful, swollen, bruised heel that the patient cannot bear weight on; the heel may look broadened and shortened with the normal medial arch flattened. Watch for fracture blisters and compartment syndrome of the foot.
Böhler's angle on the lateral radiograph; flattening below ~20° indicates depression of the posterior facet. Investigations
Lateral and axial (Harris) radiographs of the heel. Measure Böhler's angle (normal 20–40°; reduced in depression fractures) and Gissane's angle. CT is essential for intra-articular fractures to plan surgery (Sanders classification).
Management
Undisplaced / extra-articular fractures are treated conservatively — rest, elevation, no weight-bearing initially, then a period in a cast/boot with early ankle and subtalar movement. Displaced intra-articular fractures may be treated by open reduction and internal fixation to restore the joint surface, heel height and width, though management is debated and wound complications are common; primary subtalar arthrodesis is an option for severe comminution. Surgery is delayed until swelling settles (positive ‘wrinkle test’).
🔑KEY POINTS TO REMEMBER- Most common tarsal fracture; mechanism = axial load (fall onto heels).
- Always exclude spine, hip, tibial plateau and contralateral heel injuries.
- Böhler's angle (normal 20–40°) reduced in depression; CT for intra-articular planning.
- Undisplaced → conservative; displaced intra-articular → ORIF once swelling settles.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition & Mechanism
A tibial plateau fracture involves the articular surface of the proximal tibia. It results from an axial load combined with a valgus (or varus) force — e.g. a car bumper striking the lateral side of the knee (‘bumper fracture’) or a fall from height. Because it is intra-articular, accurate reduction is essential to avoid arthritis.
Clinical Features
A swollen, painful knee with a haemarthrosis and inability to bear weight. Assess for associated ligament injury (collaterals, cruciates), meniscal tears, neurovascular injury (popliteal artery, common peroneal nerve, especially in high-energy medial/bicondylar fractures) and compartment syndrome.
Schatzker Pattern I Lateral plateau — pure split (wedge) II Lateral plateau — split + depression III Lateral plateau — pure depression IV Medial plateau fracture (high-energy) V Bicondylar (both plateaus) VI Plateau # with metaphyseal–diaphyseal dissociation 💡Schatzker IV–VI are high-energy injuries — actively look for neurovascular damage, compartment syndrome and the ‘floating knee’; a fracture-dislocation of the knee can occlude the popliteal artery.Investigations
AP and lateral radiographs; CT is standard to define articular depression and comminution and to plan surgery. MRI if ligamentous/meniscal injury is suspected.
Management
Undisplaced or minimally depressed fractures (articular step < ~2–3 mm) may be treated in a hinged brace with protected weight-bearing and early movement. Displaced/depressed fractures need open (or arthroscopically assisted) reduction, elevation of the depressed segment, bone grafting of the defect and buttress-plate fixation to restore a congruent joint surface and the mechanical axis. High-energy V/VI patterns may be temporised with a spanning external fixator until soft tissues recover.
Complications
Early: compartment syndrome and popliteal vascular injury (high-energy IV–VI patterns), and wound problems. Late: post-traumatic osteoarthritis from an incongruent joint or residual axial malalignment, knee stiffness, and instability from associated ligament injury.
Look for associated ligament and meniscal injury. 🔑KEY POINTS TO REMEMBER- Intra-articular proximal tibia; axial + valgus/varus force (bumper injury).
- Schatzker I–III lateral, IV medial, V bicondylar, VI with shaft dissociation.
- CT for planning; exclude popliteal artery / peroneal nerve injury & compartment syndrome.
- Undisplaced → brace; displaced → reduction, graft the defect, buttress plating.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition
Acute compartment syndrome is a surgical emergency in which raised pressure within a closed osteofascial compartment reduces capillary perfusion below the level needed for tissue viability, causing ischaemic injury to muscle and nerve. Common sites are the leg and forearm; common causes are fractures (tibial shaft, supracondylar humerus), crush injury, tight casts, reperfusion, and burns.
Pathophysiology
Bleeding/oedema in a rigid compartment raises interstitial pressure → venous outflow obstruction → further rise in pressure → arteriolar/capillary collapse → tissue ischaemia. A vicious cycle ensues; irreversible muscle necrosis begins within 4–6 hours, leading eventually to Volkmann's ischaemic contracture.
Clinical Features — the 5 Ps (with a crucial caveat)
The earliest and most reliable sign is pain out of proportion to the injury, aggravated by passive stretching of the muscles in the compartment. Paraesthesia follows. Pallor, pulselessness and paralysis are late and unreliable — the presence of a distal pulse does not exclude compartment syndrome, because the compartment pressure rarely exceeds systolic arterial pressure.
⚠️Do not wait for pulselessness or paralysis, and do not rely on a palpable pulse for reassurance. Remove all encircling casts/dressings and re-assess. If suspicion persists, measure compartment pressure (ΔP = diastolic − compartment pressure < 30 mmHg is an indication to decompress).Sites, Causes & a Note on the Chronic Form
Common compartments affected are the anterior and deep posterior of the leg and the volar forearm. Causes include fractures (tibial shaft, supracondylar humerus), crush and reperfusion injury, tight plaster casts or dressings, prolonged limb compression, burns and bleeding disorders. Note the entirely different chronic exertional compartment syndrome: activity-related pain in athletes that resolves with rest, diagnosed by dynamic pressure testing and treated electively (not an emergency).
Management
This is a time-critical emergency. Remove constricting casts and bandages, keep the limb at heart level (not elevated, which lowers perfusion), give oxygen and analgesia, and correct hypotension. The definitive treatment is immediate open fasciotomy of all compartments (a two-incision, four-compartment release in the leg), left open and closed later (delayed primary closure or skin graft). Necrotic muscle is debrided; watch for rhabdomyolysis and acute kidney injury.
Diagnosis is clinical — do not wait for pressure measurement. 🔑KEY POINTS TO REMEMBER- Emergency: raised compartment pressure → ischaemia of muscle and nerve.
- Pain out of proportion + pain on passive stretch = earliest signs.
- Pulses present until late — never exclude it on the basis of a palpable pulse.
- Treatment = urgent open fasciotomy of all compartments; watch for rhabdomyolysis.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition
Fat embolism syndrome (FES) is a clinical condition in which fat globules enter the circulation and lodge in the pulmonary and systemic capillaries, causing a characteristic triad of respiratory, cerebral and cutaneous features. It classically follows fractures of long bones and the pelvis (especially the femoral shaft) in young adults, typically 24–72 hours after injury.
Pathogenesis
Two theories are combined: the mechanical theory (marrow fat released from the fracture enters torn venous sinusoids and embolises to the lungs) and the biochemical theory (free fatty acids generated from these globules cause a toxic vasculitis and ARDS-like alveolar damage).
Clinical Features (Gurd's criteria)
Major criteria: respiratory insufficiency (tachypnoea, hypoxia, ARDS), cerebral involvement (confusion, drowsiness, restlessness) and a petechial rash (over the conjunctivae, axillae, neck and chest). Minor criteria include fever, tachycardia, retinal changes, fat in the urine/sputum, thrombocytopenia and a falling haematocrit. The petechial rash is the most specific sign.
💡Suspect FES in a young patient who becomes hypoxic, confused and develops petechiae 1–3 days after a femoral or tibial shaft fracture.Investigations
The diagnosis is clinical (Gurd's criteria); no single test is confirmatory. Supportive findings include hypoxaemia on arterial blood gas (a falling PaO₂ is often the earliest objective clue), thrombocytopenia and anaemia, patchy bilateral infiltrates on the chest radiograph (‘snowstorm’ appearance), and fat globules in the urine or sputum. A high index of suspicion in the at-risk patient is more valuable than any test.
Management
Treatment is largely preventive and supportive. Prevention: adequate resuscitation, early splintage and early operative stabilisation of long-bone fractures reduce the incidence. Supportive care is the mainstay — oxygen to maintain saturation, and ventilatory support (CPAP/mechanical ventilation) for respiratory failure, with fluid balance and general intensive care. Steroids have a debated prophylactic role. Most patients recover fully with timely supportive care.
Early fracture fixation reduces the incidence. 🔑KEY POINTS TO REMEMBER- Follows long-bone/pelvic fractures, 24–72 h later, in young adults.
- Triad: respiratory (hypoxia/ARDS), cerebral (confusion) and petechial rash.
- Gurd's criteria; petechiae are the most specific feature.
- Prevent with early fracture stabilisation; treat supportively with oxygen/ventilation.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition & Importance
A fracture of the neck of the talus results from a violent dorsiflexion force (classically the ‘aviator's astragalus’ from the rudder pedal in a crash; now more often an RTA or fall). Like the femoral head and scaphoid, the talus has a tenuous, largely retrograde blood supply and no muscle attachments, so displaced fractures carry a high risk of avascular necrosis of the body.
Clinical Features & Investigations
There is pain, swelling and inability to bear weight, with obvious deformity in displaced fracture-dislocations and, frequently, tented or threatened skin that may progress to necrosis. AP, lateral and Canale-view radiographs demonstrate the neck fracture; the Canale view best profiles the talar neck for reduction. CT defines comminution and any associated body or process fractures and confirms joint congruity.
Hawkins type Displacement AVN risk I Undisplaced neck fracture Low (~10%) II Subtalar joint subluxation/dislocation Moderate (~40%) III Subtalar + ankle joint dislocation High (~90%) IV III + talonavicular dislocation Very high 💡The Hawkins sign — a subchondral radiolucent band in the talar dome at 6–8 weeks — indicates preserved vascularity (bone resorption requires blood flow) and is a good prognostic sign; its absence suggests AVN.Management
Undisplaced (Hawkins I) fractures are treated in a non-weight-bearing cast. Displaced fractures (II–IV) are an emergency requiring urgent reduction (to relieve pressure on skin and vessels) followed by open reduction and internal fixation with screws to restore anatomy and maximise the chance of union and revascularisation. Prolonged non-weight-bearing follows.
⚠️A displaced talar neck fracture-dislocation is a surgical emergency — delayed reduction increases skin necrosis and the already high risk of avascular necrosis.Hawkins sign (subchondral lucency) indicates preserved vascularity. 🔑KEY POINTS TO REMEMBER- Dorsiflexion injury; talus has a tenuous retrograde blood supply → high AVN risk.
- Hawkins I–IV: rising displacement = rising AVN risk (I ~10% to III ~90%).
- Hawkins sign (subchondral lucency at 6–8 wk) indicates preserved vascularity.
- Undisplaced → NWB cast; displaced → emergency reduction + ORIF.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.Definition
A pilon (plafond) fracture is a fracture of the distal tibial articular surface (the plafond) caused by high-energy axial loading that drives the talus up into the tibia (a fall from height or RTA). It is distinct from a rotational ankle fracture: there is severe articular comminution and marked soft-tissue injury. (‘Pilon’ = pestle, the talus acting as a pestle against the tibial mortar.)
Clinical Features
A grossly swollen, deformed distal leg after a high-energy injury, often with fracture blisters and threatened or open skin. Assess the neurovascular status and screen for associated axial injuries (calcaneus, tibial plateau, spine). The key contrast is with a rotational ankle fracture: a pilon results from axial compression (talus driven upward), so it damages the weight-bearing articular surface with metaphyseal impaction and far greater soft-tissue injury, whereas a rotational fracture spares the plafond and is comparatively low-energy. This difference dominates both management and prognosis.
Investigations
AP, lateral and mortise radiographs and, essentially, a CT scan to define the articular fragments and plan reconstruction (Ruedi-Allgower classification).
Management
Because the soft-tissue envelope is so badly injured, management is usually staged: initial spanning external fixation (‘travelling traction’) to restore length and protect the skin, then delayed definitive ORIF once swelling subsides and the skin wrinkles. The goals are an anatomically reduced joint surface, restored alignment and stable fixation. Severe open or non-reconstructable injuries may need definitive external fixation or, late, ankle arthrodesis.
Classification & Complications
The Ruedi–Allgöwer classification grades the injury by articular displacement and comminution (I undisplaced, II displaced with moderate comminution, III severely comminuted). Pilon fractures are notorious for complications: wound breakdown and deep infection (the dominant early problem, driven by the soft-tissue injury), stiffness, post-traumatic osteoarthritis of the ankle from articular damage, malunion and non-union. Outcomes correlate strongly with the initial soft-tissue and articular injury.
⚠️Early definitive plating through swollen, blistered skin risks catastrophic wound breakdown and infection — respect the soft tissues and stage the surgery.Soft tissue condition dictates the timing of definitive fixation. 🔑KEY POINTS TO REMEMBER- High-energy axial injury of the distal tibial articular surface (plafond).
- Severe articular comminution + major soft-tissue injury (fracture blisters).
- CT is essential for planning.
- Stage it: span with external fixator first, then delayed ORIF once soft tissues recover.
📚SOURCES: Maheshwari's Essential Orthopaedics; Apley & Solomon's System of Orthopaedics and Trauma; AO Principles of Fracture Management.